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ABOUT COLLECTION NEWSLETTER ABOUT X-force 1000 Followers WORKS WITH Autodesk Autocad Architecture Workbench Autodesk AutoCAD P&ID Metheus Meshmixer Plantform T-spline CADTutor Softwr MaxonCellular and molecular responses in the gene-targeted mouse brain: Age-dependent modifications of the neuronal nitric oxide synthase. Nitric oxide (NO) mediates numerous intracellular actions in the central nervous system, and NO synthase (NOS) in the brain has been identified as a neuronal NO synthase (nNOS) in rodents. The nNOS gene is localized on chromosome 5q34-35, and several alternatively spliced nNOS transcripts have been detected. We previously showed that the nNOS+/+ mice brains possess higher NO-related amino acid levels as well as NOS activity compared to those of nNOS-/- mice. Here, we report age-dependent modifications in nNOS+/+ mouse brains. At 5 months, which is the initial stage of the life cycle, no marked differences were observed between nNOS+/+ and nNOS-/- mouse brains. At 12 months, the level of nitrotyrosine (a marker of peroxynitrite generated by NO and superoxide anion) in the nNOS+/+ mouse brain was significantly increased compared with that of the nNOS-/- mouse brain. Furthermore, the nNOS+/+ mouse brain exhibited considerably lower levels of alpha-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA), an excitatory neurotransmitter, compared to those of the nNOS-/- mouse brain. In contrast, at 12 months, the level of kainic acid (a selective neurotoxic agonist for the AMPA/kainate receptors), a putative marker of AMPA-mediated excitotoxic responses, was significantly lower in the nNOS-/- mouse brain compared to that of the nNOS+/+ mouse brain. Immunoblot analysis revealed that the level of AMPA receptor subunit GluR1 in nNOS-/- mouse brains was lower than that in nNOS+/+ mouse brains.